1, The full form of HGF is Human Growth Foundation. It’s used on Medical ,Paediatrics & Child Health in United States

The Human Growth Foundation (HGF) is the oldest nonprofit organization of its kind. HGF serves as a global leader in endocrine research, education, patient advocacy, and support.

​HGF provides:

• Research Support
• Family Education and Advocacy
• Public Outreach & Awareness
• Support of Parents of Children with Growth Disorders
• Support of Adults with Growth Hormone Deficiency
• Training for Growth Specialists
• Education for Healthcare Providers

The Human Growth Foundation (HGF) was founded in 1965, by five families of children with growth disorders. HGF’s primary purpose was to identify other parents and children and to seek the support of the public for research and treatment for

Their vision is to help children with growth disorders, adults with growth hormone deficiency, and healthcare providers reach new heights by providing awareness/outreach programs. HGF intends to offer support, features, and up-to-date information on newly discovered and existing disorders, diseases, and syndromes of growth abnormalities, in an effort to increase awareness and the importance of early diagnosis and treatment.growth hormone deficiency.

2, The full form of HGF is Hepatocyte Growth Factor. It’s used on Medical ,Anatomy & Physiology in Worldwide

Hepatocyte growth factor (HGF) or scatter factor (SF) is a paracrine cellular growth, motility and morphogenic factor. It is secreted by mesenchymal cells and targets and acts primarily upon epithelial cells and endothelial cells, but also acts on haemopoietic progenitor cells and T cells. It has been shown to have a major role in embryonic organ development, specifically in myogenesis, in adult organ regeneration, and in wound healing

It has been more than 25 years since HGF was discovered as a mitogen of hepatocytes. HGF is produced by stromal cells, and stimulates epithelial cell proliferation, motility, morphogenesis and angiogenesis in various organs via tyrosine phosphorylation of its receptor, c-Met. In fetal stages, HGF-neutralization, or c-Met gene destruction, leads to hypoplasia of many organs, indicating that HGF signals are essential for organ development. Endogenous HGF is required for self-repair of injured livers, kidneys, lungs and so on. In addition, HGF exerts protective effects on epithelial and non-epithelial organs (including the heart and brain) via anti-apoptotic and anti-inflammatory signals. During organ diseases, plasma HGF levels significantly increased, while anti-HGF antibody infusion accelerated tissue destruction in rodents. Thus, endogenous HGF is required for minimization of diseases, while insufficient production of HGF leads to organ failure. This is the reason why HGF supplementation produces therapeutic outcomes under pathological conditions. Moreover, emerging studies delineated key roles of HGF during tumor metastasis, while HGF-antagonism leads to anti-tumor outcomes. Taken together, HGF-based molecules, including HGF-variants, HGF-fragments and c-Met-binders are available as regenerative or anti-tumor drugs. Molecular analysis of the HGF-c-Met system could provide bridges between basic biology and clinical medicine.